Multiple Myeloma an incurable disease, but I have spent the last 25 years in remission using a blend of conventional oncology and evidence-based nutrition, supplementation, and lifestyle therapies from peer-reviewed studies that your oncologist probably hasn't told you about.
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Multiple myeloma (MM) patients are often told that their cancer “is incurable but very treatable.” When I was first diagnosed with multiple myeloma I found this statement to be confusing. When I found a study showing that cannabidiol kills myeloma and enhances Velcade, I understood how MM could be incurable but treatable.
The study linked and excerpted below is an excellent example of how multiple myeloma is incurable but treatable. Your oncologist will admit that Velcade (bortezomib) will eventually stop working. But cannabidiol is cytotoxic to MM and it enhances the efficacy of Velcade.
Cannabidiol causes your MM to be more sensitive to Velcade. More importantly, there are many other evidence-based, non-toxic therapies that also make your MM more sensitive to Velcade.
Myeloma is about two things:
MMers usually don’t die from their cancer. They die from health problems caused by their cancer such as bone damage and/or from the toxicity caused by chemotherapy and radiation.
The solution? Take an integrative approach to managing your multiple myeloma. Enhance the efficacy of chemotherapy and while you reduce the toxicity. I live an anti-MM lifestyle based on nutrition, supplementation, lifestyle, bone health and mind-body therapies.
If you’d like to learn more about the following topics from an evidence-based perspective, please click the blue button below to register for a FREE webinar:
“Because bortezomib (BORT) (Velcade) is commonly used in MM treatment, we investigated the effects of Cannabidiol and BORT in CD138+TRPV2- MM cells and in MM cell lines transfected with TRPV2 (CD138+TRPV2+).
These results showed that Cannabidiol by itself or in synergy with BORT strongly inhibited growth, arrested cell cycle progression and induced MM cells death by regulating the ERK, AKT and NF-κB pathways with major effects in TRPV2+ cells. These data provide a rationale for using Cannabidiol to increase the activity of proteasome inhibitors in MM.”