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Colorectal Cancer Non-Conventional Therapies-

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Colorectal cancer has been shown to respond to many antioxidant supplements before, during and after diagnosis.

Image result for photo of colon cancer

Depending on your age, stage at diagnosis and your prognosis and therapy plan nutrition, supplementation and other lifestyle therapies can both enhance conventional therapies and/or kill colon cancer on their own.

The four studies linked and excerpted below are a few of the many evidence-based but non-conventional (not FDA approved) therapies that colorectal cancer patients and survivors should consider when learning about their colorectal cancer diagnosis and prognosis.

I am a long-term cancer survivor and cancer coach. I was diagnosed with my blood cancer in 1994 and was told “we can do nothing more for you” in September of 1997 after exhausting all conventional therapies for my cancer. I underwent a non-conventional cancer therapy called Antineoplaston Therapy from 11/97-4/99 and have been in complete remission from my “incurable cancer” ever since.

 I keep my cancer in complete remission by living an evidence-based. non-toxic, lifestyle through nutrition, supplementation, bone health, etc. therapies.

Have you been diagnosed with colorectal cancer? Scroll down the page, post a question or comment and I will reply to you ASAP.

Thank you,

David Emerson

  • Cancer Survivor
  • Cancer Coach
  • Director PeopleBeatingCancer

Recommended Reading:


Vitamin D and Colon Cancer

“”They found 107 systematic literature reviews, 74 meta-analyses of observational studies of plasma vitamin D concentrations, and 87 meta-analyses of randomized controlled trials of vitamin D — a huge body of literature that has been summarized and reviewed repeatedly, with a whole variety of nicely methodologically and statistically strong results. Where does it take us?
The role of vitamin D in colorectal cancer prevention, and possibly in treatment, is biologically plausible.

A vast literature has been summarized many times over. The conclusion of this umbrella review was that in cancer, particularly colorectal cancer, the evidence is suggestive of benefit for vitamin D…”

Antioxidants enhance the cytotoxicity of chemotherapeutic agents in colorectal cancer: a p53-independent induction of p21WAF1/CIP1 via C/EBPbeta.

“Colorectal cancer (CRC) is the second leading cause of cancer deaths in the United States. Five-fluorouracil (5FU) remains the single most effective treatment for advanced disease, despite a response rate of only 20%. Herein, we show that the antioxidants pyrrolidinedithiocarbamate and vitamin E induce apoptosis in CRC cells. This effect is mediated by induction of p21WAF1/CIP1, a powerful inhibitor of the cell cycle, through a mechanism involving C/EBPbeta (a member of the CCAAT/enhancer binding protein family of transcription factors), independent of p53. Antioxidants significantly enhance CRC tumor growth inhibition by cytotoxic chemotherapy in vitro (5FU and doxorubicin) and in vivo (5FU). Thus, chemotherapeutic agents administered in the presence of antioxidants may provide a novel therapy for colorectal cancer.

Melatonin potentiates flavone-induced apoptosis in human colon cancer cells by increasing the level of glycolytic end product

“Melatonin is a natural compound synthesized by a variety of organs. It has been described to possess cell protecting activity in normal cells but was shown to induce apoptotic cell death in cancer cells. We determined to which extent and based on which molecular mechanisms melatonin is able to cause apoptosis in HT-29 human colon cancer cells…

Our study provides evidence that melatonin potentiates flavone-induced apoptosis in HT-29 human colon cancer cells by enhancing the level of oxidizable substrates that can be transported into mitochondria in the presence of flavone.”

Alpha-Lipoic acid induces apoptosis in human colon cancer cells by increasing mitochondrial respiration with a concomitant O2-*-generation.

“In conclusion, our study provides evidence that ALA and DHLA can effectively induce apoptosis in human colon cancer cells by a prooxidant mechanism that is initiated by an increased uptake of oxidizable substrates into mitochondria.”

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