Regardless of type and stage of your head and neck cancer diagnosis you will want to consider evidence-based, non-toxic, inexpensive therapy that studies show is cytotoxic to head and neck cancer.
I am both a cancer survivor and cancer coach. Surviving head and neck cancer is about researching and using the best of both conventional (FDA approved) and evidence-based, non-conventional therapies. Grape Seed Extract is an example of an evidence-based, non-toxic head and neck cancer therapy.
If it was possible to patient Grape Seed Extract, it would be a wonder drug:
I take Life Extension Grape Seed Extract because of its formula and efficacy.
Have you been diagnosed with head and neck cancer? What type? What stage and symptoms if any? Please scroll down the page, post a question or comment and I will reply to you ASAP.
“A study published this week in the journal Carcinogenesis shows that in both cell lines and mouse models, grape seed extract (GSE) kills head and neck squamous cell carcinoma cells, while leaving healthy cells unharmed…
Grape seed extract creates these conditions that are unfavorable to growth. Specifically, the paper shows that grape seed extract both damages cancer cells’ DNA (via increased reactive oxygen species) and stops the pathways that allow repair (as seen by decreased levels of the DNA repair molecules Brca1 and Rad51 and DNA repair foci)…
The Agarwal Lab hopes to move in the direction of clinical trials of grape seed extract, potentially as an addition to second-line therapies that target head and neck squamous cell carcinoma that has failed a first treatment…”
“The aim of this study is to examine the antioral cancer effects of different concentrations of GSE in terms of cell viability, apoptosis, reactive oxygen species (ROS), mitochondrial function, and DNA damage…
Results-High concentrations (50–400 μg/ml) of GSE dose-responsively inhibited proliferation of oral cancer Ca9-22 cells but low concentrations (1–10 μg/ml) of GSE showed a mild effect in a MTS assay. For apoptosis analyses, subG1 population and annexin V intensity in high concentrations of GSE-treated Ca9-22 cells was increased but less so at low concentrations…
Additionally, high concentrations of GSE dose-responsively induced more γH2AX-based DNA damage than low concentrations.
Conclusions- Differential concentrations of GSE may have a differentially antiproliferative function against oral cancer cells via differential apoptosis, oxidative stress and DNA damage.”