Multiple Myeloma an incurable disease, but I have spent the last 25 years in remission using a blend of conventional oncology and evidence-based nutrition, supplementation, and lifestyle therapies from peer-reviewed studies that your oncologist probably hasn't told you about.
Click the orange button to the right to learn more about what you can start doing today.
Genetically speaking, multiple myeloma is more complicated than many cancers. That may sound like a tall statement to make for someone who does not have a degree in genetics but hear me out. Like all cancers, MM is caused by uncontrolled cell growth. Fine. But according to studies, MMers face other genetic challenges:
According to the last study linked below, people who are genetically pre-disposed to heart disease can dramatically reduce their risk of heart attack and stroke by improving their cardiorespiratory fitness.
I would be the first person to say that heart health is child’s play next to treating MM. But my reasoning is about epigenetics, not disease. In other words, like heart health, regardless of genetic challenges, epigenetics can be a therapy for MMers.
I’m not saying that non-conventional therapies will cure your MM. I’m saying that evidence-based therapies, those proven to fight cancer, are an essential complementary therapy for MM patients and survivors. These therapies can change your genetic expression.
If our genetic expression can cause our MM, maybe changing our genetic expression can heal our incurable blood cancer.
In the post The Ultimate Cancer Therapy – Eight Slices of the Magic Bullet I made a case for evidence-based non-toxic therapies to reduce the risks of cancer. I think it is reasonable to suggest that these eight non-toxic therapies cause genes to express themselves differently.
Many MMers supplement with curcumin. Research has demonstrated that curcumin changes the genetic espression in MMers. I’m simply extending this logic to include anti-MM nutrition, supplementation, exercise, detox, etc.
I don’t believe my MM remission since 1999 is a coincidence. I don’t believe that the Burzynski therapy called antineoplaston therapy (ANP) cured me. I believe that I can relapse any time like all MMers. I believe that I have to work to remain in complete remission from my MM.
To complicate matters, I am also genetically pre-disposed to heart disease. I live with chemotherapy-induced heart damage and though I have never been diagnosed Marfan’s Syndrome runs in my family.
“We are more than the sum of our genes. Epigenetic mechanisms modulated by environmental cues such as diet, disease or our lifestyle take a major role in regulating the DNA by switching genes on and off…
In our body we find more than 250 different cell types. They all contain the exact same DNA bases in exactly the same order; however, liver or nerve cells look very different and have different skills. What makes the difference is a process called epigenetics. Epigenetic modifications label specific regions of the DNA to attract or keep away proteins that activate genes. Thus, these modifications create, step by step, the typical patterns of active and inactive DNA sequences for each cell type. Moreover, contrary to the fixed sequence of ‘letters’ in our DNA, epigenetic marks can also change throughout our life and in response to our environment or lifestyle. For example, smoking changes the epigenetic makeup of lung cells, eventually leading to cancer. Other influences of external stimuli like stress, disease or diet are also supposed to be stored in the epigenetic memory of cells.
“With the development of molecular techniques (microarrays and next-generation sequencing), our understanding has been highly improved in the past 5 years. These studies have not only confirmed the prevalence of wide heterogeneity in myeloma at the molecular level, but has also provided a much clearer picture of the disease pathogenesis and progression…
Despite the use of the most promising genomic tools (GEP, SNP array, and NGS), MM remains a very heterogeneous disease, with no unique common mutation. The NGS studies in the past 5 years have characterized the suclonality concept. Even though NGS data can be considered disappointing because they do not show common mutations that could define subentities, they are nevertheless important because they confirm the wide molecular heterogeneity of the disease and the frequent occurrence of some supposedly “driver” mutations only in subclones…”
“In one of the largest observational studies on fitness and heart disease, researchers examined data collected from nearly a half-million people in the UK Biobank database. They found that people with higher levels of grip strength, physical activity and cardiorespiratory fitness had reduced risks of heart attacks and stroke, even if they had a genetic predisposition for heart disease…
Among those considered at high genetic risk for heart disease, high levels ofwere associated with a 49 percent lower risk for coronary heart disease and a 60 percent lower risk for atrial fibrillation compared with study participants with low cardiorespiratory fitness…