Non-Toxic Prostate Cancer Therapy

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” Epidemiologic studies indicate that selenium is a potential prostate cancer preventive and decreases the growth rate of prostate cancer cells.”

You’ve been diagnosed with early stage prostate cancer. Your Gleason score is 6. You are trying to decide between active surveillance or treatment. Evidence-based, non-toxic therapies have been shown to reduce your risk of progression without the side effects of treatment. Treat your early stage prostate cancer with evidence-based, non-toxic therapies.

Or to put it a different way, “treatment” of your prostate cancer risk does not have to lead to urinary incontinence or other difficult side effects.

Numerous studies cite your ability to reduce your risk of prostate cancer through diet, supplementation and lifestyle therapies.

Illustration Of Prostate

There are a host of evidence-based, non-conventional therapies that research has shown kill PCa. The studies linked and excerpted below cite how two well-studied, non-toxic, non-conventional supplements, curcumin and ginger, kill prostate cancer.

I am a long-term survivor of an incurable cancer and a cancer coach. I have remained in complete remission from my cancer by living an evidence-based, non-toxic, anti-cancer lifestyle through nutrition, supplementation, lifestyle and more.

For more information about managing your prostate cancer, scroll down the page, post a question and I will reply ASAP.

David Emerson

  • Cancer Survivor,
  • Cancer Coach,
  • Director PeopleBeatingCancer

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What is the role of selenium in prostate cancer risk-reduction?

“Selenium is an essential, nonmetallic trace element that is widely distributed throughout the body. It is a component of multiple antioxidant enzymes and participates in various functions. Epidemiologic studies indicate that selenium is a potential prostate cancer preventive and decreases the growth rate of prostate cancer cells. Plasma, serum, and tissue levels of selenium are inversely associated with the risk of developing prostate cancer.

Selenium is found in Brazil nuts, walnuts, fish (including canned tuna and shellfish), beef, turkey, chicken, eggs, whole grains, garlic, onions, broccoli, cabbage, and mushrooms. One of the problems in obtaining adequate dietary selenium is that the level of selenium in a given plant depends on the soil in which it is growing. For example, produce from the Imperial Valley in California has a higher selenium content than plants grown elsewhere.

Benefits of whole ginger extract in prostate cancer.

” Ginger has been known to display anti-inflammatory, antioxidant and antiproliferative activities, indicating its promising role as a chemopreventive agent.

Here, we show that whole ginger extract (GE) exerts significant growth-inhibitory and death-inductory effects in a spectrum of prostate cancer cells. Comprehensive studies have confirmed that GE perturbed cell-cycle progression, impaired reproductive capacity, modulated cell-cycle and apoptosis regulatory molecules and induced a caspase-driven, mitochondrially mediated apoptosis in human prostate cancer cells…”

Curcumin inhibits cancer-associated fibroblast-driven prostate cancer invasion through MAOA/mTOR/HIF-1α signaling.

“However, curcumin abrogated CAF-induced invasion and EMT, and inhibited ROS production and CXCR4 and IL-6 receptor expression in prostate cancer cells through inhibiting MAOA/mTOR/HIF-1α signaling, thereby supporting the therapeutic effect of curcumin in prostate cancer…”

Green tea and the risk of prostate cancer

“Prostate cancer (PCa) now remains the 2nd most frequently diagnosed cancer. In recent years, chemoprevention for PCa becomes a possible concept. Especially, many phytochemicals rich foods are suggested to lower the risk of cancer.

Among these foods, green tea is considered as effective prevention for various cancers. However, clinical trials and previous meta-analyses on the relationship between green tea consumption and the risk of PCa have produced inconsistent outcomes.

This study aims to determine the dose–response association of green tea intake with PCa risk and the preventive effect of green tea catechins on PCa risk.

Seven observational studies and 3 randomized controlled trials were retrieved from

  • Cochrane Library,
  • PubMed,
  • Sciencedirect Online,
  • and hand searching.

The STATA (version 12.0) was applied to analyze the data. The relative risks (RRs) and 95% confidence intervals were pooled by fixed or random effect modeling.

Dose–response relations were evaluated with categories of green tea intake. Although there was no statistical significance in the comparison of the highest versus lowest category, there was a trend of reduced incidence of PCa with each 1 cup/day increase of green tea (P = 0.08).

Our dose–response meta-analysis further demonstrated that higher green tea consumption was linearly associated with a reduced risk of PCa with more than 7 cups/day. In addition, green tea catechins were effective for preventing PCa with an RR of 0.38 (P = 0.02).

In conclusion, our dose–response meta-analysis evaluated the association of green tea intake with PCa risk systematically and quantitatively. And this is the first meta-analysis of green tea catechins consumption and PCa incidence. Our novel data demonstrated that higher green tea consumption was linearly reduced PCa risk with more than 7 cups/day and green tea catechins were effective for preventing PCa. However, further studies are required to substantiate these conclusions.”

Exercise and prostate cancer

“Prostate cancer is a leading cause of cancer morbidity and mortality in men. In addition to improved treatments, strategies to reduce disease risk are urgently required. This review summarises the literature that examines the association between exercise and prostate cancer risk.

  • Between 1989 and 2001, 13 cohort studies were conducted in the US and internationally. Of these, nine showed an association between exercise and decreased prostate cancer risk.
  • Five of 11 case-control studies conducted between 1988 and 2002 reported an association between decreased risk of prostate cancer and high activity levels.

Considering all studies performed between 1976 and 2002, 16 out of 27 studies reported reduced risk in men who were most active; in nine out of 16 studies the reduction in risk was statistically significant. Average risk reduction ranged from 10-30%.

In aggregate, this evidence suggests a probable link between increased physical exercise and decreased prostate cancer risk. The ability of exercise to modulate hormone levels, prevent obesity, enhance immune function and reduce oxidative stress have all been postulated as mechanisms that may underlie the protective effect of exercise. Exercise may also be of benefit in men undergoing treatment for prostate cancer.

Overall, study design and control of potential confounding factors varied greatly among studies, possibly contributing to the variation in results. Epidemiological studies that are better controlled, larger in scale and more carefully designed may help to more fully clarify the relationship between exercise and prostate cancer. In addition, intervention trials that test whether exercise programmes can reduce prostate cancer risk are currently underway to rigorously test the ability of exercise to reduce prostate cancer incidence.”


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