Curcumin, Integrative Therapies, Can Overcome Multidrug-Resistance In Lung Cancer

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Integrative Therapies Can Enhance the Efficacy of Conventional Lung Cancer (LC) Chemotherapy while Reducing the Risk Of Side Effects

The newly diagnosed lung cancer patient has several challenges facing him/her. It is common knowledge that LC is one of the most aggressive cancers with dismal five-year survival rate. But it is important to dig into why five -year LC survival rates are so dismal. In short:

According to the study linked and excerpted below curcumin causes apoptosis in LC and can overcome multi-drug resistance from conventional LC chemotherapy. I have remained in complete remission from a different but equally difficult cancer since 1999 by living an evidence-based, non-conventional lifestyle through nutrition, supplementation, etc.

To learn more about evidence-based therapies for lung cancer, please watch the short video below:


Have you been diagnosed with lung cancer? What stage? What therapies are you considering? Scroll down the page, post a question or comment and I will reply to you ASAP.

Thank you,

David Emerson

  • Cancer Survivor
  • Cancer Coach
  • Director PeopleBeatingCancer

Recommended Reading:

Curcumin promotes apoptosis in A549/DDP multidrug-resistant human lung adenocarcinoma cells through an miRNA signaling pathway.

Curcumin extracted from the rhizomes of Curcuma longa L. has been shown to have inhibitory effects on cancers through its anti-proliferative and pro-apoptotic activities.

Emerging evidence demonstrates that curcumin can overcome drug resistance to classical chemotherapies.

In our study, we first demonstrated that curcumin had anti-cancer effects on A549/DDP multidrug-resistant human lung adenocarcinoma cells.”

Green tea polyphenol EGCG suppresses lung cancer cell growth through upregulating miR-210 expression caused by stabilizing HIF-1α.

” In summary, our results demonstrated, for the first time, the elevation of miR-210 by EGCG in LC cell lines and this is mediated by the stabilization of HIF-1α. This event contributes to the anticancer activity of EGCG….”

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